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Alzheimer’s Disease
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Insights Into Low Molecular Weight and Cytotoxic Aggregates from in Vitro and Computer Experiments - Molecular Basis o
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Artikel-Nr:
9781848167544
Veröffentl:
2012
Einband:
HC gerader Rücken kaschiert
Erscheinungsdatum:
31.12.2012
Seiten:
466
Autor:
Philippe Derreumaux
Gewicht:
822 g
Format:
235x157x29 mm
Sprache:
Englisch
Beschreibung:
Alzheimer's disease is the most common form of senile dementia, affecting more than 24 million people worldwide. It is characterised pathologically by abnormally high levels of brain lesions (senile plaques) in dead and dying neurons, and by elevated numbers of amyloid deposits in the walls of cerebral blood vessels. The major component of senile plaques is a small protein of 39 43 amino acids called amyloid- (A ). Thus far, no treatment has been shown to slow the progression of sporadic and familial Alzheimer's disease.
Cellular and In Vitro Aspects of Amyloid-Beta (Aβ) Aggregation: Amyloid Hypothesis, Molecular and cellular Aspect of Toxicity (R Kayed et al.); Biophysical Characterization of Aβ Aggregation (D Teplow et al.); Coordination of Metal Ions to Aβ Peptide: Impact on AD (P Faller et al.); Inhibitor Design Against Cytotoxic Aβ Species (A Doig et al.); Bioinformatics and Computer Simulations of Aβ Aggregation Under Various Environmental Conditions: Predicting Amyloid-Prone Fragments from Amino Acid Sequences (F Chiti et al.); Kinetics of Amyloid Growth (N Mousseau et al.); Chain Growth Fibril using All-Atom MD Simulations (D Klimov et al.); Investigating the Interaction Between Oligomers and Membranes (Y Mu et al.); Molecular Insights into the Assembly of Aβ on Surfaces and Nanotubes (G Wei et al.); and other papers.